Publications of Susanne Lucae
All genres
Conference Paper (4)
2017
Conference Paper
42 (Suppl. 1), T26, pp. S310 - S311 (2017)
Aging- and Stress-Related Epigenetic Disinhibition of FKBP5 Contributes to NF-KB-Driven Inflammation and Cardiovascular Risk. 56th Annual Meeting of the American-College-of-Neuropsychopharmacology (ACNP), Palm Springs, CA, December 03, 2017 - December 07, 2017. NEUROPSYCHOPHARMACOLOGY Meeting Abstract (5)
2025
Meeting Abstract
28, p. i314 - i314. (2025)
GENOMIC AND IMMUNOLOGICAL BIOMARKERS FOR PREDICTING TREATMENT OUTCOME IN DEPRESSION. In INTERNATIONAL JOURNAL OF NEUROPSYCHOPHARMACOLOGY, 2014
Meeting Abstract
17, RA-01-003, p. 42 - 42. 29th CINP World Congress of Neuropsychopharmacology
, Vancouver, Canada, June 22, 2014 - June 26, 2014. Oxford University Press, Oxford, UK (2014)
Genetic predictor of antidepressant response for major depressive disorder accounting for clinical subgroups: A genome-wide association study. In INTERNATIONAL JOURNAL OF NEUROPSYCHOPHARMACOLOGY,
Meeting Abstract
122 (3), LB15. 57th Symposium of the German Society of Endocrinology, Dresden, March 19, 2014 - March 22, 2014. Johann Ambrosius Barth Verlag Medizinverlage Heidelberg GmbH, Stuttgart, Germany (2014)
IGF-1 levels and polymorphisms in the IGF-1 receptor are associated with response to antidepressant treatment. In EXPERIMENTAL AND CLINICAL ENDOCRINOLOGY & DIABETES,
Meeting Abstract
75 (9), p. 45S - 45S. 69th Annual Scientific Convention and Meeting of the
Society-of-Biological-Psychiatry, New York, NY, May 08, 2014 - May 10, 2014. Elsevier Science Inc., New York, NY, USA (2014)
RNA Expression Profiling in Age-/Severity-Matched Depressed Inpatients Revealed New Candidates as Possible Biomarkers for Antidepressant Response. In BIOLOGICAL PSYCHIATRY, 2013
Meeting Abstract
73 (9), 747, p. 239S - 239S. 68th Annual Scientific Meeting of the Society-of-Biological-Psychiatry, San Francisco, CA, May 16, 2013 - May 18, 2013. (2013)
Recurrent Depression is Associated with Earlier Ocurrence of Metabolic Syndrome and Aggravates the Effect of Vascular Risk Factors on Cerebral Microangiopathy Determined By MRI. In Biological Psychiatry,