Publikationen von Susanne Lucae
Alle Typen
Konferenzbeitrag (4)
201.
Konferenzbeitrag
42 (Suppl. 1), T26, S. S310 - S311 (2017)
Aging- and Stress-Related Epigenetic Disinhibition of FKBP5 Contributes to NF-KB-Driven Inflammation and Cardiovascular Risk. 56th Annual Meeting of the American-College-of-Neuropsychopharmacology (ACNP), Palm Springs, CA, 03. Dezember 2017 - 07. Dezember 2017. NEUROPSYCHOPHARMACOLOGY Meeting Abstract (5)
202.
Meeting Abstract
28, S. i314 - i314. (2025)
GENOMIC AND IMMUNOLOGICAL BIOMARKERS FOR PREDICTING TREATMENT OUTCOME IN DEPRESSION. In INTERNATIONAL JOURNAL OF NEUROPSYCHOPHARMACOLOGY, 203.
Meeting Abstract
17, RA-01-003, S. 42 - 42. 29th CINP World Congress of Neuropsychopharmacology
, Vancouver, Canada, 22. Juni 2014 - 26. Juni 2014. Oxford University Press, Oxford, UK (2014)
Genetic predictor of antidepressant response for major depressive disorder accounting for clinical subgroups: A genome-wide association study. In INTERNATIONAL JOURNAL OF NEUROPSYCHOPHARMACOLOGY, 204.
Meeting Abstract
122 (3), LB15. 57th Symposium of the German Society of Endocrinology, Dresden, 19. März 2014 - 22. März 2014. Johann Ambrosius Barth Verlag Medizinverlage Heidelberg GmbH, Stuttgart, Germany (2014)
IGF-1 levels and polymorphisms in the IGF-1 receptor are associated with response to antidepressant treatment. In EXPERIMENTAL AND CLINICAL ENDOCRINOLOGY & DIABETES, 205.
Meeting Abstract
75 (9), S. 45S - 45S. 69th Annual Scientific Convention and Meeting of the
Society-of-Biological-Psychiatry, New York, NY, 08. Mai 2014 - 10. Mai 2014. Elsevier Science Inc., New York, NY, USA (2014)
RNA Expression Profiling in Age-/Severity-Matched Depressed Inpatients Revealed New Candidates as Possible Biomarkers for Antidepressant Response. In BIOLOGICAL PSYCHIATRY, 206.
Meeting Abstract
73 (9), 747, S. 239S - 239S. 68th Annual Scientific Meeting of the Society-of-Biological-Psychiatry, San Francisco, CA, 16. Mai 2013 - 18. Mai 2013. (2013)
Recurrent Depression is Associated with Earlier Ocurrence of Metabolic Syndrome and Aggravates the Effect of Vascular Risk Factors on Cerebral Microangiopathy Determined By MRI. In Biological Psychiatry,